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group or order unique events). Programs based on a conceptualist stance hold that nonscientists and scientists alike hold conceptualizations of reality. The history of science teaches that common-sense conceptualizations can be improved and ultimately replaced with scienti cally honed ones. And programs based on a formalist position suggest possible alternative universal primitives (such as feel, good, and bad ) and bolster our claim that emotion is a heterogeneous cluster of events. CORE AFFECT AS A POINT OF DEPARTURE Next, we search for primitive entities. One reason that basic emotions are ill suited to serve as emotion primitives has been established by research from the basic emotions perspective: They are too complex. For example, they typically consist of separable components (Izard, 1977) and are directed at an object (i.e., one fears, loves, hates, or is angry with something). Oatley and Johnson-Laird (1987) pointed out that an emotional primitive should be free of this something (the object) because of the cognitive involvement that the object implies. Curiously, then, our search for emotional primitives begins with moods and other simple feelings that lack an object. In this way, Oatley and Johnson-Laird created an important new theory based on a categorical perspective. Here we explore that same approach but from a dimensional perspective. The goal in dimensional studies is to nd what is common to various emotions, moods, and related states. Methods have included multivariate analyses of selfreported feelings, introspection, the semantic differential, and various biological techniques. This research has regularly found such broad dimensions as pleasure-displeasure and activation-deactivation. We refer to any state that can be de ned simply as some combination of these two dimensions as core affect.
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out a selected course of action. Managers control the implemented course of action to ensure the plan is being followed and, as necessary, modified to meet the objectives of the selected course of action.
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Mandler, G. (1975). Mind and emotion. New York: Wiley. Mandler, G. (1979). Emotion. In E. Hearst (Ed.), The rst century of experimental psychology (pp. 275 321). Hillsdale, NJ: Erlbaum. Mandler, G. (1984). Mind and body: Psychology of emotion and stress. New York: Norton. Mandler, G. (1990). A constructivist theory of emotion. In N. S. Stein, B. L. Leventhal, & T. Trabasso (Eds.), Psychological and biological approaches to emotion (pp. 21 43). Hillsdale, NJ: Erlbaum. Mandler, G. (1992). Emotions, evolution, and aggression: Myths and conjectures. In K. T. Strongman (Ed.), International Review of Studies on Emotion (pp. 97 116). Chichester, England: Wiley. Mandler, G. (1993). Thought, memory, and learning: Effects of emotional stress. In L. Goldberger & S. Breznitz (Eds.), Handbook of stress (pp. 40 55). New York: Free Press. Mandler, G. (1999). Emotion. In B. M. Bly & D. E. Rumelhart (Eds.), Cognitive science (pp. 367 384). San Diego, CA: Academic Press. Mandler, G., & Kremen, I. (1958). Autonomic feedback: A correlational study. Journal of Personality, 26, 388 399. Mandler, G., Mandler, J. M., & Uviller, E. T. (1958). Autonomic feedback: The perception of autonomic activity. Journal of Abnormal and Social Psychology, 56, 367 373. Mandler, G., & Watson, D. L. (1966). Anxiety and the interruption of behavior. In C. D. Spielberger (Ed.), Anxiety and behavior (pp. 263 288). New York: Academic Press. Mara on, G. (1924). Contribution l tude de l action emotive de l adr naline. Revue fran aise d Endocrinologie, 2, 301 325. McCosh, J. (1880). The emotions. New York: Scribner. McNaughton, M. (1989). Biology and emotion. Cambridge, England: Cambridge University Press. Meyer, L. B. (1956). Emotion and meaning in music. Chicago: University of Chicago Press. Mowrer, O. H. (1939). Stimulus-response analysis of anxiety and its role as a reinforcing agent. Psychological Review, 46, 553 565. Ortony, A., Clore, G. L., & Collins, A. (1988). The cognitive structure of emotions. New York: Cambridge University Press. Ortony, A., & Turner, T. J. (1990). What s basic about basic emotions Psychological Review, 97, 315 331. Patkai, P. (1971). Catecholamine excretion in pleasant and unpleasant situations. Acta Psychologia, 35, 352 363. Paulhan, F. (1887). Les ph nom nes affectifs et les lois de leur apparition. Paris: F. Alcan. Paulhan, F. (1930). The laws of feeling. London: Kegan Paul, Trench, Trubner. Pick, J. (1970). The autonomic nervous system. Philadelphia: Lippincott. Reymert, M. L. (1928). Feelings and emotions: The Wittenberg Symposium. Worcester, MA: Clark University Press. Reymert, M. L. (1950). Feelings and emotions: The Mooseheart Symposium. New York: McGraw-Hill.
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Underneath the Value column, there are numerous blank spaces. To ll in these blanks with information that identi es the name of the musician, the title of the album, the year the album was made, and so forth, simply click one of the blanks and type the relevant data. When you nish editing the tag, click the Apply button. Exit the tag by clicking the OK button.
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Step B: Pre-Rinse with Water
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ANIMAL MODELS OF NICOTINE ADDICTION Animal models examining the reinforcing effects of nicotine have been used to assess the various contributing factors of tobacco dependence as observed in the human population. The extent to which animal models can be used to interpret the underlying nature of dependence in humans depends mainly on the validity of the model. Animal models have been evaluated based on predictive, face, and construct validity (Willner, 1991). Predictive validity of an animal model is de ned as performance in the test predicts performance in the condition being modeled. For example, valid animal models of drug reward can differentiate between drugs that are abused by humans and those that are not and can therefore be used to evaluate whether a novel drug possesses abuse liability as well as to detect potential candidate medications for prevention of drug addiction. Face validity is an indication of whether the behavioral and pharmacological qualities of an animal model are similar in nature to those seen in the human condition. Construct validity is assessed by determining whether there is a sound theoretical rationale between the animal model and the human condition being modeled (Willner, 1991). Table 7.1 addresses the questions that assess the validity of each animal model discussed next as related to nicotine addiction. Several animal models have been used to examine the reinforcing effects of nicotine. In the following paragraphs, we discuss methodology, ndings directly related to nicotine addiction, and validity (see Table 7.1) of two frequently used animal models, the self-administration and the placeconditioning paradigms. Self-Administration The self-administration (SA) paradigm provides a measure of the reinforcing effects of drugs. The animal learns the relationship of its behavior such as pressing a lever or a nosepoke and a reinforcer such as an i.v. injection of a drug. If the relationship between the animal s behavior and the response is reinforcing, the probability of the animal continuing the behavior is increased. It has taken 10 to 15 years of research with animals to map out the conditions that will support reliable SA of nicotine. Nicotine SA has been demonstrated in nonhuman primates (Goldberg, Spealman, & Goldberg, 1981), rats (Corrigall & Coen, 1989; Donny, Caggiula, Knopf, & Brown, 1995), and mice (Picciotto et al., 1998; Stolerman, Naylor, Elmer, & Goldberg, 1999). The role of the mesocorticolimbic dopamine system in mediating nicotine SA has also been examined. For example, lesions of dopaminergic neurons in the NAc, and administration of
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Activate any worksheet, and select File Save As. Then select Microsoft Excel Addin (*.xla) from the Save as type drop-down list.
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