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Note that running init q does not start or stop System V services. It is used mostly when tinkering with the gettys defined in inittab. To manage services, you can use the chkconfig and service commands. For example, to start the Samba service immediately, you could type this:
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Tyler, C.R., B. van der Erden, S. Jobling, T. Panter & J.P. Sumpter (1996) Measurement of vitellogenin, a biomarker for exposure to oestrogenic chemicals, in a wide variety of cyprinid sh. Journal of Comparative Physiology B-Biochemical Systemic and Environmental Physiology, 166, 418 426. Tyler, C.R., R. van Aerle, T.H. Hutchinson, S. Maddix & H. Trip (1999a) An in vivo testing system for endocrine disruptors in sh early life stages using induction of vitellogenin. Environmental Toxicology and Chemistry, 18 (2), 337 347. Tyler, C.R., T. Hutchinson, J. Harries, K. Thorpe, S. Maddix & J. Sumpter (1999b) Development of in vivo testing systems for endocrine disrupting chemicals in sh (Abstract 2j/001). 9th Annual Meeting of SETAC-Europe, Leipzig, Germany, 25 29 May 1999. USEPA (1986) Fish Life-Cycle Toxicity Tests. US Environmental Protection Agency, Hazard Evaluation Division, Standard Evaluation Procedure, USA, EPA 540/9-86-137, 11 pp. van der Kraak, G.J., K.R. Munkittrick, M.E. McMaster, C.B. Portt & J.P. Chang (1992) Exposure to bleached kraft mill ef uent disrupts the pituitary-gonadal axis of white sucker at multiple sites. Toxicology and Applied Pharmacology, 115, 224 233. van der Kraak, G., K.R. Munkittrick, M.E. McMaster, & D.L. MacLatchy (1998) A comparison of bleached kraft mill ef uent, 17 -estradiol, and -sitosterol effects on reproductive function in sh. In (eds Kendall, R.J., R.L. Dickerson, J.P. Giesy, & W.A. Suk) Principles and Processes for Evaluating Endocrine Disruption in Wildlife. SETAC Technical Publication, SETAC Press, Pensacola, pp. 249 265. van Look, K.J.W. & D.E. Kime (1999) Fish sperm motility as a monitor of reproductive disruption by heavy metals (Abstract PP-138). 6th International Symposium on Reproductive Physiology of Fish, 4 9 July 1999, Bergen, Norway. von Westernhagen, H., H. Rosenthalm, V. Dethlefsen, W. Ernst, U. Harms & P.-D. Hansen (1981) Bioaccumulating substances and reproductive success in Baltic ounder Platichthys esus. Aquatic Toxicology, 1, 85 99. von Westernhagen, H., P. Cameron, V. Dethlefsen & D. Janssen (1989) Chlorinated hydrocarbons in North Sea whiting (Merlangius merlangus), and effects on reproduction. 1. tissue burden and hatching success. Helgol nder Meeresuntersuchungen, 43, 45 60. Walthall, W.K. & J.D. Stark (1997) A comparison of acute mortality and population growth rate as endpoint of toxicological effect. Ecotoxicology and Environmental Safety, 37, 45 52. Weisberg, S.B., P. Himchak, T. Baum, H.T. Wilson & R. Allen (1996) Temporal trends in abundance of sh in the tidal Delaware River. Estuaries, 19 (3), 723 729. Wertheimer, A.C. & A.G. Celewycz (1996) Abundance and growth of juvenile pink salmon in oiled and non-oiled locations of western Prince William Sound after the Exxon Valdez oil spill. American Fisheries Society Symposium, 18, 518 532. Wester, P.W. (1991) Histopathological effects of environmental pollutants b-HCH and methyl mercury on reproductive organs of freshwater sh. Comparative Biochemistry and Physiology, 100C, 237 239. Wester, P.W. & J.H. Canton (1986) Histopathological study of Oryzias latipes (Medaka) after longterm b-hexachlorocyclohexane exposure. Aquatic Toxicology, 9, 21 45. Westin, D.T., C.E. Olney & B.A. Rogers (1985) Effects of parental and dietary organochlorines on survival and body burdens of striped bass larvae. Transactions of the American Fisheries Society, 114, 125 136. Whipple, J., M. Eldridge, P. Benville, M. Bowers, B. Harvis & N. Stapp (1981) The effects of inherent parental factors on gamete condition and variability in striped bass (Morone saxatilis). Rapports et Proc s-verbaux de R unions du Conseil International pour l Exploration de la Mer, 178, 93 94.
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The prolonged survival of patients with impaired immunity is becoming more and more commonplace. The number of patients on cytotoxic drugs and steroids for the treatment of malignant disease, and to suppress immunity in connective tissue disorders and after organ transplantation, is increasing. The incidence and prevalence of AIDS are also increasing. These immunosuppressed or immunode cient patients are susceptible to infections (Fig. 15.5): by organisms which are capable of causing infection in normal individuals, but which cause abnormally frequent and severe infections in the immunocompromised; by organisms which are not pathogenic in normal circumstances, so-called opportunistic infections. The clinical features of these infections are often ill-de ned and not distinct from the patient s underlying disease. The different organisms do not create diagnostic clinical syndromes. Intensive investigation, in close collaboration with the microbiology laboratory, is usually required to establish the diagnosis and the correct treatment.
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This, however, can be true only when all patents in the portfolio were issued on the same date and will expire on the same date in the future. In real life this rarely happens. To overcome this problem, we first note that
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