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Failures should be transparent to users; otherwise, theywill be unsatisfied with the system. If failures of servers are not transparent to designers of client applications, they have to build measures into their clients to achieve failure transparencyfor their users. This will complicate the design of clients and make them more expensive and difficult to maintain.
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A lter generator is a running key generator for stream cipher applications. It consists of a single LFSR that is ltered by a nonlinear Boolean function f . This model has been in practical use for generating the keystream of a stream cipher. However, the strength of this model depends on the choice of the nonlinear Boolean function (Fig.13.2). 13.2.8 Synchronous and Asynchronous Stream Ciphers
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Unfortunately, solving such an equation for large N is extremely tedious. See Winkler and Schwartz (1972) for an alternative solution. The paper lists quadratic ratios for directivity and Q, for the uniform case, optimum directivity, and optimum directivity for a prescribed Q. They also compared several end re cases, as discussed in Section See Section 2.4.1 for additional bandwidth data. Kovacs and Solymar (1956) treated the inverse problem: Given directivity for an array con guration, nd the excitation that minimizes Q. They showed that the minimum Q increases as D2 for a xed array size.
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Released March 20, 2002. As we prepare for our possible IPO it is necessary to apply wherever possible the principles of corporate governance found in the best practices of public companies. This will allow our shareholders, members, potential investors, employees, and the public at large to clearly understand and support the Exchange. With less than one month before our Annual Shareholders meeting, and in light of the present environment of increased scrutiny of for-pro t companies, I would like to highlight several outstanding issues that need to be resolved as soon as possible for us to continue to make progress on this front. With respect to the work of the Audit Committee I understand that among the issues you are analyzing are several that involve corporate governance (e.g., the need for committee charters, including decision rights and responsibilities) as well as how to determine and resolve con icts of interest regarding topics brought to committees and the Board. Given our unique nature as the rst major U.S. exchange to demutualize, and the two-year transition envisioned in the original S-4 registration statement, the nuances associated with some of these issues could not have been anticipated prior to now. Nonetheless, they are of paramount importance today, and the Board needs the bene t of analysis by your Committee and outside counsel in order to determine the best course of action to be taken by the Exchange. I encourage you to proceed without delay. As to issues within the purview of the Compensation Committee (on which I serve), there has been much discussion and analysis over the past year regarding how best to compensate Board members, many of whom dedicate countless hours in assisting the Board and management. For the most part, the Committee s work in the area of determining fair and equitable cash compensation paid to Directors has been completed, and the Board 314
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Most PET studies of eating disorders have evaluated glucose metabolism (metabolic rate, blood ow) with the use of 18- uorodeoxyglucose (FDG). At rest with eyes closed, patients with anorexia nervosa usually present an absolute global and regional hypometabolism of glucose. However, this can also be found in low-weight patients with depression and is most likely a consequence of starvation since it disappears after weight gain. Most recently regional cerebral blood ow (rCBF) was investigated using PET in 9 women in long-term recovery from BN (Frank et al., 2000). There were no differences between bulimics and controls, suggesting that alterations in rCBF during the ill state of bulimia nervosa may be a state-related phenomenon. Both, anorexic and normal-weight bulimic patients have been shown to have relative parietal hypometabolism which persisted after weight gain in some anorexic patients (Delvenne et al., 1995, 1997a, 1997b, 1999). The authors hypothesise that this might be a characteristic of eating disorders, a primary cerebral dysfunction, which may be related to distortions of body image. However, it might also be a particular sensitivity of this cortical region to nutritional factors. In their most recent study Delvenne et al. (1999) found that patients with anorexia nervosa showed an increased relative glucose metabolism in the inferior frontal cortex and in the basal ganglia compared to controls, con rming the nding by Herholz et al. (1987). This might be due to a more pronounced reduction in cortical than in basal ganglia glucose metabolism and may be related to increased vigilance or anxiety in patients compared to controls. Two studies of glucose metabolism in bulimia applied PET scans together with cognitive activation tasks which stimulate particular regions of brain activity (Wu et al., 1990; Andreason et al., 1992). Bulimics showed an abnormal hemispheric lateralization (left greater than right hemispheric asymmetry as opposed to healthy controls in whom right is greater than left) in the parietotemporal region and parts of the frontal lobes, which appeared to be independent of the mood state (similar ndings were reported for depression and OCD). It is an open question whether the loss of normal right activation in some areas has a role in the aetiology of this disorder. Another option is symptom provocation studies, which have already been performed in patients with anxiety disorders. In healthy subjects the confrontation with desirable food
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