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Three boundary styles affect how islands are hatched: Normal: Hatches alternating areas so that the outer area is hatched, the next inner island is not hatched, the next inner island is hatched, and so on.
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lthough Linux is developed around an ideal of sharing and trust, the world is not always that kind. In this chapter you take a look at the various types of threats to your identity, data, and property and then review some practical steps you can take to neutralize them. The chapter includes extensive coverage of GNU Privacy Guard, an application that provides industrial-strength encryption for data. Finally, I cover some resources for learning more about security issues.
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FIGURE 12.26. Couder two-element compensator.
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(1) (1) (1) (1) pn (xk ) = f (x0 )L0 (xk ) + f (x1 )L1 (xk ) + f (x2 )L2 (xk ).
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References: Al, Fe: life cycle inventories for packagings. Environmental series no. 250/I. Swiss agency for the environment, forset and landscape. Berne, 1998 Plastics: actual data sets of APME (http:// lca.apme.org. date: 31.05.2000)
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In the last chapter, we encountered two instances of resonant behavior, both stated in terms of field imperfections. If there are steering errors, however small, an integer value of the tune will lead to an oscillation growing in amplitude linearly from turn to turn without bound. The without bound is of course an artifact of our approximations, but because of the limited aperture in which the particle motion must be accommodated, the approximations are reasonable. In the second case, it was found that at a tune such that 2 v = m where m is an integer, any quadrupole error will lead to amplitude growth. We did not prove it then, but the growth in this case can be faster than linear. In this section, we will attempt to generalize the discussion of resonances to include nonlinear field perturbations.
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In the Live Enabler Options section, you can decide when you want to check for object enablers. Object enablers let the user display and use custom objects in drawings, even when the application that created them is unavailable. For example, if you send a customer who is using Release 2000 a drawing with associative dimensions, that customer can use an object enabler to view those dimensions.
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humans complexed with other high molecular weight glycoproteins such as 2-macroglobulin and thrombospondin-1; the latter are involved in regulating in ammation and tissue remodelling (Wang et al., 2006), and may interfere with adiponectin s bioavailability. The HMW form is the most closely related to insulin resistance and the metabolic syndrome (Hara et al., 2006). Serum HMW complexes correlate better with glucose tolerance than total adiponectin levels (Fisher et al., 2005; Lara-Castro et al., 2006; Aso et al., 2006), and HMW levels increase selectively when insulin sensitivity improves in type 2 diabetic patients treated with TZDs (Pajvani et al., 2004). Moreover, relative increases in HMW and MMW isoforms, and a reduction in LMW adiponectin, occur after weight reduction (Bobbert et al., 2005). Two types of adiponectin receptor, encoded on different chromosomes, have been described (Yamauchi et al., 2003). Both AdipoR1 and AdipoR2 are 7-transmembrane receptors, with the atypical feature of the N-terminus lying on the inside and the C-terminus outside the membrane; both can form homo- or hetero-multimers. In humans, AdipoR1 is ubiquitously expressed, with highest levels in skeletal muscle, whereas AdipoR2 is predominantly expressed in liver. Little is known about their regulation in humans (Kadowaki et al., 2006; Staiger et al., 2004). Exercise training up-regulates AdipoR1 and AdipoR2 expression in human skeletal muscle (Bl her et al., 2006), while no signi cant change is reported in subcutaneous adipose tissue during energy restriction (Viguerie et al., 2005). An adiponectin receptor interacting protein, APPL1, has recently been identi ed (Mao et al., 2006). This 70-amino acid protein occurs in insulin-sensitive tissues that also express adiponectin receptors; APPL1 binds to adiponectin receptors and enhances downstream responses such as fatty acid oxidation, GLUT4 translocation and glucose uptake, AMPK protein levels and phosphorylation. Unlike leptin and other adipokines, circulating levels of adiponectin are inversely related to BMI and body fat mass (Matsubara, Maruoka and Katayose, 2002) (Figure 4.13). This relationship is interesting, in light of the evidence that adiponectin enhances insulin sensitivity and protects against atherogenesis, and suggests a potential link between obesity and its cardio-metabolic risks. Adiponectin may confer bene cial metabolic and cardiovascular
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some worryingly big questions remain unanswered, mostly because there are no adequate long-term data on ef cacy, safety and costeffectiveness. Is it justi able to use these drugs for longer than a couple of years, especially in young people who could potentially face life-long treatment If not, does a limited course of pharmacotherapy buy enough lasting bene ts by reducing the risks of morbidity or premature death, or by improving quality of life Some basic physiological and pharmacological issues also need to be resolved. Weight loss with all anti-obesity drugs attens off after several months; is this due to physiological adaptation (such as the inappropriate activation of protective anti-starvation responses) or simply to poor compliance Indeed, could poor compliance explain why most patients fail to lose signi cant amounts of weight with all the drugs that have been tested Adherence to treatment for some diseases that are widely seen as dangerous is surprisingly low; for example, many type 1 diabetic patients, at risk of unpleasant hyperglycaemic symptoms and potentially life-threatening ketoacidotic coma, may deliberately omit insulin or take small and ineffective dosages. It seems reasonable to blame the lack of success of obesity management programmes on the patient s presumed inability to comply with lifestyle changes, but we can only do this if we can reject the hypothesis that anti-obesity drugs fail simply because they are not taken. Bariatric surgery has now emerged from its indifferent and poorly-appreciated infancy and stands out as the only one-off treatment that can have lasting and potentially life-long effects, and that can reduce weight suf ciently to shift some of the most persistent burdens of obesity. The reversal of type 2 diabetes is an outstandingly important clinical outcome of bariatric surgery. This nding also suggests that it may be possible, at least early in the development of the disease, to rewind the clock of -cell dysfunction although end-stage -cell fallout is irreversible and remains beyond rescue, even by bariatric surgery. The surgical procedures in current use are safer than their predecessors but are still not without risk. Moreover, bene ts are not permanent in a sizeable minority of cases and as with anti-obesity drugs, it will be important to determine whether this long-term failure is due to physiological adaptation or to non-compliance. Some aspects
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The purpose of manufacturing process validation is to ensure that the manufacturing process for the drug product is fully controlled and capable of providing a product that complies with established quality speci cations consistently and repetitively. This validation process covers manufacturing operations from formulation through packaging. Process validation is being conducted in support of launch of the drug product. Process validation also establishes documented evidence that the manufacturing process, when executed according to the VMP and pertinent process SOPs, consis-
18 Applying GoLive s JavaScripts: Actions and Smart Objects
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